Cold season why acute myocardial infarction (ami)

The first hospital affiliated to Harbin medical university, director of cardiovascular internal medicine TianYe 

Source: health

        Studies confirm that the temperature and cardiovascular disease mortality in u-shaped relation, every 1 ℃ below the temperature of cold region, cardiovascular disease mortality rate increases by 1%. There are statistics show that, during Christmas time, westerners more than 4.65% of cardiac deaths and more than 4.99% of the occurrence of cardiac death. Foreign scholars pointed out that the drop in atmospheric temperature 10 ℃, 13% of the total increase the incidence of coronary events, coronary heart disease events and death rate increases by 11%. When the average daily temperature is lower than 26.2 degrees Celsius, acute myocardial infarction (mi) admission rate will increase by 30% ~ 70%. Cold cold northern areas of China because of its special climate, for the high incidence of cardiovascular system diseases. As winter temperatures below minus 19 degrees Celsius in Harbin in China, the death toll with the decreasing temperature increased significantly, 2.7% of the population died can be attributed to the low temperature cold, especially in patients with coronary heart disease (CHD) is the biggest impact low temperature cold.

        In recent years, the cold stress in acute myocardial infarction pathological physiology and pathogenesis of attention and influence more and more get the attention of academic circles. In summary, the cold stress can be induced by the sympathetic nervous excitement, aggravate atherosclerotic inflammation, cause endothelial dysfunction, adaptive thermogenesis increase ATP production and relevant mechanism and cause of acute myocardial infarction (mi), become the important driving force behind the scenes of the four acute myocardial infarction.

        Animal experiments showed that the cold exposed rats could activate the body's sympathetic adrenergic system, make its circulation, urinary system of epinephrine and norepinephrine levels increased significantly, which in turn leads to increased blood pressure, platelet aggregation and blood viscosity increased; Strong and last spasm or coronary artery coronary artery vascular intimal injury, hemorrhage, and acute thrombosis, can lead to acute coronary artery occlusion and acute myocardial infarction.

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